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两种甘扩展蛋白编码基因有助于胃口孔隙,与对甘的结构性抵抗有关

Zongling Liu^1,2, Zhuoxin Yu¹, Xiufang Li¹

¹State Key Laboratory for Conservation and Utilization of Subtropical Agro-Bioresources, College of Life Science and Technology, Guangxi University, Nanning 530004, China.

Journal of fungi (Basel, Switzerland)

|September 27, 2024

概括

甘污垢耐药性涉及结构因素,特别是口腔特征. 某些Expansin基因会影响口腔孔径,为繁殖抗病甘品种提供潜力.

科学领域:

植物病理学植物病理学
分子生物学分子生物学
农业学是一种农业学.

背景情况:

甘污垢,由*Sporisorium scitamineum*引起,显著降低了作物产量.
抵抗机制包括结构性,生化和生理因素,结构性抵抗尚未得到研究.
胃口在植物病原体相互作用和疾病发展中发挥着至关重要的作用.

研究的目的:

调查甘中结构性抗性的作用.
识别影响口腔特征的遗传因素,与污垢耐药性相关.
探索甘Expansin基因在疾病耐药性繁殖中的潜力.

主要方法:

根据不同的注射方法对甘品种 (ZZ9和GT42) 的比较分析.
评估耐药和易感品种的口腔孔径和密度.
甘芽的RNA测序用于识别和表征Expansin基因.

主要成果:

甘品种ZZ9表现出对污垢的结构性耐药性.
易受雾影响的品种 (ROC22,GT42) 的口腔口径和密度高于耐药品种 (ZZ1,ZZ6,ZZ9).
已经确定了两种甘Expansin基因 (c111037.graph_c0和c113583.graph_c0) 增加了口腔孔径.

结论:

甘口腔,特别是它们的孔径和密度,是对污垢结构抵抗的关键组成部分.
已识别的Expansin基因有助于口腔调节,并代表增强甘污垢耐药性的潜在目标.
这些发现为通过基因改进开发抗病甘提供了洞察力.

关键词:

扩展蛋白质是一种扩展蛋白质.在RNA-seqqq.电阻的电阻是指电阻的电阻胃的胃,就是胃的胃.甘的污秽是因为甘的污秽.

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