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Updated: Jul 13, 2026

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慢性双S (BPS) 暴露在阿波利波蛋白E缺陷小鼠中的亲有风源效应和潜在机制

Ying-Bing Zuo1, Zeng-Jin Wen2, Meng-Die Cheng1

  • 1Institute for Translational Medicine, The Affiliated Hospital of Qingdao University, College of Medicine, Qingdao University, Deng Zhou Road 38, Qingdao 266021, China; Department of Cardiology, The Affiliated Hospital of Qingdao University, Qingdao University, 16 Jiangsu Road, Qingdao 266000, China.

Ecotoxicology and environmental safety
|September 29, 2024
PubMed
概括

双S (BPS) 暴露会通过促进炎症和细胞粘附,使小鼠的动脉样硬化 (AS) 恶化. 这项研究揭示了BPS是心血管疾病进展的危险因素.

关键词:
双 S 是一种双醇.动脉样硬化 动脉样硬化内皮细胞是内皮细胞.巨细胞是一个巨细胞.血管光滑肌肉细胞细胞

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