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Updated: Jun 11, 2025

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免疫反应基因1的缺陷通过诱导M1巨细胞两极分化和加剧Ly6C高单细胞招募来加剧由炎症引发的心脏功能障碍

Song Shen1, Jianhui Li1, Zhonghai Wei1

  • 1Department of Cardiology of Nanjing Drum Tower Hospital, The Affiliated Hospital of Nanjing University Medical School, Nanjing, China.

Biology direct
|October 1, 2024
PubMed
概括

IRG1/itaconate通路在败血症期间保护心脏. 伊塔科纳特治疗通过激活NRF2/HO-1通路来保护心脏功能并减少炎症.

关键词:
伊他康酸盐是什么意思巨细胞的两极分化单细胞动员活动.这就是NRF2的NRF2.败血性心肌损伤 败血性心肌损伤

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