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Updated: Aug 13, 2026

High Resolution Electron Microscopy of the Helicobacter pylori Cag Type IV Secretion System Pili Produced in Varying Conditions of Iron Availability
09:05

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Published on: November 21, 2014

由Helicobacter pylori感染引起的细胞恶性特征的GRB7介导增强

Huilin Zhao1,2, Si Chen1,3, Xinfeng Bai4

  • 1Department of Pathogenic Biology, School of Basic Medical Sciences, Binzhou Medical University, Yantai, China.

Frontiers in microbiology
|October 3, 2024
PubMed
概括

杆菌感染通过STAT3激活在胃癌细胞中的增长因子受体结合蛋白7 (GRB7) 上调. 这种GRB7过度表达通过激活ERK信号来促进癌症的进展,提供治疗点.

关键词:
这就是CagAA.GRB7 GRB7 GRB7 GRB7 GRB7 GRB7 GRB7 GRB7 GRB7 GRB7 GRB7 GRB7在Helicobacter Pylori的研究中,在STAT3中,我们可以使用STAT3.胃癌 胃癌 是一种胃癌.

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