校正:甲状腺激素释放激素对小脑长期抑郁和运动学习的贡献
Masashi Watanave1, Yasunori Matsuzaki1, Yasuyo Nakajima2
1Department of Neurophysiology and Neural Repair, Gunma University Graduate School of Medicine, Maebashi, Japan.
Frontiers in cellular neuroscience
|October 11, 2024
概括
这项研究纠正了上一篇文章DOI. 修正后的DOI确保了科学作品的准确引用和检索.
科学领域:
- 神经科学是一个神经科学.
- 细胞生物学 细胞生物学
背景情况:
- 确保准确的科学记录保存对于研究完整性至关重要.
- 适当的引用有助于获取和验证科学发现.
研究的目的:
- 纠正之前发表的文章的数字物体标识符 (DOI).
- 维护科学文献的准确性和可追溯性.
主要方法:
- 发布了一份纠正通知.
- 对该物品提供了正确的DOI.
主要成果:
- 这篇文章的DOI已更新为10.3389/fncel.2018.00490.
- 这确保读者能够访问预期的研究.
结论:
- 准确的DOI对科学界至关重要.
- 这次纠正符合科学出版的标准.
更多相关视频
相关概念视频
Long-term Depression
2.5K
Long-term depression, or LTD, is one of the ways by which synaptic plasticity—changes in the strength of chemical synapses—can occur in the brain. LTD is the process of synaptic weakening that occurs over time between pre and postsynaptic neuronal connections. The synaptic weakening of LTD works in opposition to synaptic strengthening by long-term potentiation (LTP) and together are the main mechanisms that underlie learning and memory.
Calcium Ion Concentration Mechanism
If over...
Calcium Ion Concentration Mechanism
If over...
2.5K
Long-term Potentiation
54.9K
Long-term potentiation, or LTP, is one of the ways by which synaptic plasticity—changes in the strength of chemical synapses—can occur in the brain. LTP is the process of synaptic strengthening that occurs over time between pre- and postsynaptic neuronal connections. The synaptic strengthening of LTP works in opposition to the synaptic weakening of long-term depression (LTD) and together are the main mechanisms that underlie learning and memory.
54.9K


