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Updated: Jun 10, 2025

Vasodilation of Isolated Vessels and the Isolation of the Extracellular Matrix of Tight-skin Mice
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肌肉中的Nrf2缺乏减弱了实验性自身免疫性肌肉炎诱导的肌肉虚弱

Koichi Himori1, Mami Yamada1, Takahiro Onoki2

  • 1Graduate School of Science, Nagoya City University, Nagoya, Japan.

The Journal of physiology
|October 21, 2024
PubMed
概括

实验性自身免疫性肌肉炎 (EAM) 通过增加T淋巴细胞透和炎症性细胞因子,导致肌肉疲软. 向 肌肉中的核因素红色素2相关因子2 (Nrf2) 活性显示为治疗EAM诱导的肌肉衰弱有前途.

关键词:
这是T淋巴细胞.这是一种化学物质,chemokine.异形性炎症性肌肉病变 (Idiopathic Inflammatory Myopathy) 是一种异形性炎症性肌肉病变 (Idiopathic Inflammatory Myopathy) 是一种异形性炎症性肌肉病变.肌肉疲弱 肌肉疲弱 肌肉疲弱与核因子红色素2相关的核因子2因子

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