在患有良性前列腺增生症的患者中,脱体低活性是长期COVID的尿动力学特征吗?
Ning Xiao1,2,3, Jinhua Xiao4, Qi Tang1
1Department of Urology, Video Urodynamic Studies Center, The Second Affiliated Hospital of Guilin Medical University, Guilin, China.
Medicine
|October 28, 2024
概括
在良性前列腺增生症 (BPH) 患者中,COVID-19 感染加剧了脱体低活性 (DU). 这项研究强调了DU作为长期COVID的潜在泌尿动力学特征,影响膀收缩性.
科学领域:
- 泌尿器科 泌尿器科 泌尿器科 泌尿器科
- 传染性疾病 传染性疾病
- 老年学是指老年学的学科.
背景情况:
- 虽然COVID-19会影响多个器官,但它对泌尿系统的影响,特别是BPH患者的膀脱器收缩性,尚未得到充分研究.
- 之前的研究主要使用问卷来评估COVID-19患者下尿路症状.
研究的目的:
- 调查COVID-19对良性前列腺增生症 (BPH) 患者膀脱器收缩性的影响.
- 为了比较BPH患者与没有COVID-19病史的BPH患者之间发生的破坏器低活性 (DU) 的发生率.
主要方法:
- 进行了超声波视频尿动力学研究.
- 对BPH患者 (COVID-19组) 和没有 (非COVID-19组) 之前的COVID-19感染的临床数据进行了审查和比较.
- 分析了非侵入性参数,以查看与体缩性和DU检测的相关性.
主要成果:
- 与非COVID-19组相比,在COVID-19组中观察到显著更高的破坏器低活性 (DU) 发生率.
- 患有COVID-19之前的BPH患者表现出较低的状体收缩性和膀壁厚度增加.
- 排尿后残留的尿液在COVID-19组中显示出与detrusor收缩率的线性相关性.
结论:
- 由于膀输出阻塞,COVID-19感染似乎会在BPH患者中恶化先前存在的脱体损伤.
- 阻断器低活性 (DU) 可能是长期COVID的泌尿动力学后果.
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