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学校中的1型糖尿病:综合性文献综述
Stacey Parke1, Steven James2, Megan Paterson1
1School of Health, University of the Sunshine Coast, Petrie, Queensland 4502, Australia; Department of Paediatric Diabetes and Endocrinology, John Hunter Children's Hospital, New Lambton, New South Wales 2305, Australia.
Journal of pediatric nursing
|November 4, 2024
概括
学校工作人员需要更好的培训来管理儿童的1型糖尿病 (T1D). 目前的教育形式各不相同,这凸显了在小学中需要标准化的T1D管理支持.
科学领域:
- 儿科 儿科 儿科
- 内分泌学 在内分泌学.
- 公共卫生 公共卫生
背景情况:
- 患有1型糖尿病 (T1D) 的儿童需要在学时持续管理.
- 学校工作人员在支持患有T1D的学生方面发挥着至关重要的作用,特别是在独立性有限的小学环境中.
- 现有的文献缺乏对学校工作人员提供的糖尿病教育的全面审查.
研究的目的:
- 对学校工作人员进行1型糖尿病 (T1D) 教育的综合性文献审查.
- 识别和总结T1D管理培训提供给小学设置的现有研究.
- 突出目前为学校工作人员提供T1D教育的差距.
主要方法:
- 进行了综合性文献综述.
- 包括2013年以后以英语出版的研究,重点关注支持T1D学生的小学.
- 不包括系统评论,非经验性文章和仅抽象的记录. 质量评估使用了乔安娜·布里格斯批判性评估工具.
主要成果:
- 确定并分析了15项相关研究.
- 在接受T1D管理培训的学校工作人员数量中存在显著差异.
- 一些使用葡萄糖的学校缺乏训练有素的工作人员,数据往往不区分大都市和农村环境.
结论:
- 在学校提供T1D管理教育是不一致的.
- 需要进行进一步的研究,以探索包括农村环境在内的不同地理区域糖尿病教育的差异.
- 标准化的T1D教育,持续的支持和提高学校工作人员的意识对于最佳的学生护理至关重要.
相关概念视频
01:22Diabetes Mellitus: Overview and Type I Subtype
Diabetes mellitus is a chronic metabolic disorder characterized by high blood glucose levels due to inadequate insulin production, insulin resistance, or both. The condition affects millions worldwide and can significantly impact their health and quality of life.
Type 1 diabetes is an autoimmune disease in which the immune system mistakenly attacks and destroys the insulin-producing beta cells in the pancreas. As a result, the body is unable to produce sufficient insulin, and individuals with...
Type 1 diabetes is an autoimmune disease in which the immune system mistakenly attacks and destroys the insulin-producing beta cells in the pancreas. As a result, the body is unable to produce sufficient insulin, and individuals with...
01:26Diabetes Mellitus: Introduction
Diabetes mellitus consists of chronic metabolic disorders characterized by persistent hyperglycemia. This elevated blood glucose results from defects in insulin secretion, impaired insulin action, or both. Insulin, produced by pancreatic β-cells, is essential for maintaining glucose homeostasis by facilitating cellular glucose uptake for energy or storage. Disruptions in insulin production or function lead to glucose accumulation in the bloodstream, causing the clinical features and long-term...
01:12Type I Diabetes I: Introduction
Type 1 diabetes mellitus is a chronic metabolic disorder characterized by an absolute deficiency of insulin resulting from the autoimmune destruction of pancreatic β-cells. Although it can occur at any age, it is most commonly diagnosed in childhood, adolescence, or early adulthood. The loss of insulin production impairs cellular glucose uptake, resulting in persistent hyperglycemia and necessitating lifelong insulin therapy.Autoimmune Destruction of β-CellsThe hallmark of type 1 diabetes is an...
01:26Type I Diabetes II: Pathophysiology
Type 1 diabetes mellitus arises from an immune-mediated destruction of pancreatic β-cells, resulting in an absolute deficiency of insulin. This process develops in genetically susceptible individuals when autoimmunity, environmental exposures, and immunologic dysregulation converge to trigger a targeted attack on the insulin-producing cells of the pancreas. The β-cells are located within the islets of Langerhans and are essential for regulating blood glucose by facilitating cellular uptake of...
01:26Type II Diabetes I: Introduction
Type 2 diabetes mellitus (T2DM) is a chronic metabolic disorder characterized by insulin resistance, in which target tissues such as the liver, muscle, and adipose tissue respond poorly to insulin. It is also associated with inadequate compensatory insulin secretion, where pancreatic β-cells fail to produce sufficient insulin. Together, these abnormalities lead to persistent hyperglycemia.EtiologyT2DM develops through a complex interaction of genetic predisposition and environmental or...
01:24Type II Diabetes II: Pathophysiology
PathophysiologyType 2 diabetes mellitus (T2DM ) is a chronic metabolic disorder characterized by insulin resistance and progressive pancreatic β-cell dysfunction, leading to impaired glucose homeostasis. It results from interactions among genetic predisposition, environmental factors, and metabolic stressors, such as overnutrition and a sedentary lifestyle.Insulin Resistance and Glucose DysregulationEarly T2DM involves insulin resistance in skeletal muscle, adipose tissue, and the liver.