在氨气压力下无氧消化:关键合成相互作用和甲生成通路的灵敏性动态-一篇综述
1Civil Engineering, School of Engineering, College of Science and Engineering, University of Galway, Galway, H91 TK33, Ireland.
Journal of environmental management
|November 4, 2024
概括
蛋白质丰富基质中的高氨含量抑制了无氧消化 (AD) 的甲化. 本综述详细介绍了微生物反应和有效的策略,如生物增强,以克服对温室气体 (GHG) 缓解的氨抑制.
科学领域:
- 生物技术和生物工程 生物技术和生物工程
- 环境科学 环境科学
- 微生物学 微生物学
背景情况:
- 富含蛋白质基质的无氧消化 (AD) 提供了显著的温室气体 (GHG) 缓解潜力,但由于氨含量高而受到阻碍.
- 氨的毒性会破坏微生物群落,并抑制甲化,从而导致低于最佳的过程性能.
研究的目的:
- 在氨压力下审查关键AD过程的灵敏性动态.
- 分析微生物相互作用和新陈代谢变化,以应对高水平.
- 确定有效的策略,以减轻AD中的氨抑制.
主要方法:
- 文献综述侧重于AD过程,微生物生态学和氨抑制.
- 对关键AD过程和微生物相互作用的敏感性动态的分析.
- 无氧消化微生物群的代谢能力的概况.
主要成果:
- 富含氨的基底AD带来了风险,在热友和中友系统中能量增益有限.
- 细菌体,菌体和协同菌体是耐氨的,而蛋白质菌和动态菌体是脆弱的.
- 合成酸盐氧化在氨气压力下转移路径,影响甲基生成.
结论:
- 氨物种对甲原组产生不同的影响,影响可回收性.
- 导电材料辅助的生物增量和现场H2补充显示出对电子转移和氨压力减轻的前景.
- 需要有针对性的策略来减轻氨抑制,以有效地减少AD和温室气体.
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