疲劳对肌肉间EMG-EMG合的作用 在男性60%的1RM工作负载下进行长椅压力运动时
Lejun Wang1, Haifeng Tao1, Qing Chen1
1Sport and Health Research Center, Shanghai YangZhi Rehabilitation Hospital (Shanghai Sunshine Rehabilitation Center), Physical Education Department, Tongji University, Shanghai, China.
Frontiers in human neuroscience
|November 6, 2024
概括
在长椅压力期间的疲劳会改变肌肉协调,降低双肩三联接,同时增强骨髓三连接. 这揭示了神经肌肉的适应性,以维持力量的产生.
科学领域:
- 神经肌肉生理学 神经肌肉生理学
- 运动科学 运动科学
- 生物力学 生物力学
背景情况:
- 了解神经肌肉控制和疲劳对于有效的运动处方至关重要.
- 目前的研究缺乏关于肌肉间肌电图 (EMG) -EMG合变化在协同和对抗肌肉动作在练习,如长椅压力时的共识.
- 这项研究通过调查疲劳引起的肌肉协调在长椅压力期间的变化来弥补这一差距.
研究的目的:
- 为了研究与疲劳相关的EMG-EMG肌肉间合的变化,在长椅压力练习期间.
- 量化肌肉协调在疲劳条件下如何适应.
主要方法:
- 31名健康的男性在60%的最大一次重复 (1RM) 时进行了长椅压力,直到疲.
- 表面EMG信号从三腕 (TB),两腕 (BB),前半身 (AD),后半身 (PD) 和大胸部 (PM) 记录下来.
- 阶段同步指数 (PSI) 计算了不同EMG频段 (马:35-60 Hz) 的协同 (AD-TB,AD-PM) 和对抗 (BB-TB,AD-PD) 肌肉对.
主要成果:
- 在运动后半段,贝塔和马频段的TB,AD,PD和PM肌肉的EMG功率增加.
- 在双腕和三腕 (对手对) 之间观察到马波段PSI的显著下降.
- 相反,马波段EMG-EMG合显著增加了前腹骨和三骨之间 (协同对).
结论:
- 在长椅压力期间的疲劳导致对手肌肉 (BB-TB) 之间的神经驱动同步减少.
- 协同肌肉之间的合增加 (AD-TB) 表明力量维持的补偿机制.
- 这些肌肉间合的变化很可能是由于脊柱上调整和差异性激动剂-对抗剂肌肉疲劳造成的.
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