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Updated: Jul 10, 2026

The Hypoxic Ischemic Encephalopathy Model of Perinatal Ischemia
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[加斯特罗丁在新生小鼠中通过PI3K/AKT通路改善了缺氧缺血性脑损伤后的微质介导炎症反应]

H Zuo1, Z Duan1, Z Wang1

  • 1Department of Human Anatomy and Histology & Embryology, Faculty of Basic Medical Sciences, Kunming Medical University, 1168 West Chunrong Road, Kunming, 650500, China.

Nan fang yi ke da xue xue bao = Journal of Southern Medical University
|November 6, 2024
PubMed
概括

加斯特罗丁通过调节PI3K/AKT通路,降低了新生小鼠患有缺氧缺血性脑损伤 (HIBD) 的炎症. 这种神经保护作用涉及调节炎性细胞因子和微质激活.

关键词:
在PI3K/AKT中使用.这是一种胃口,一种饮食.缺氧 - 缺血性脑损伤炎症反应是一种炎症反应.微质细胞中的微质细胞缺乏氧气和糖的情况.

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