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在三乙烯诱导的免疫性肝损伤中,Wnt5a促进了库普费尔细胞激活

Lei Gao1, Ya-Ni Ding1, Peng-Cheng Zhou1

  • 1Department of Occupational Health and Environmental Health, School of Public Health, Anhui Medical University, Hefei, China.

Toxicology and industrial health
|November 26, 2024
PubMed
概括

三乙烯 (TCE) 暴露可以通过Wnt5a/JNK通路引起肝损伤,激活库弗弗细胞. 在小鼠模型中,抑制这种途径可以减少肝损伤.

关键词:
在C-Jun N-终端激酶.库弗弗细胞是库弗弗细胞.没有Wnt5a.肝脏损伤 肝脏损伤是什么?三乙烯是一种三乙烯.

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