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Updated: May 27, 2026

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在高葡萄糖下,由脂肪组织巨细胞衍生的外体MiR-500a-5p通过抑制Nrf2表达促进脂肪细胞炎症

Yong-Zhen Li1, Yuan Tian2, Chen Yang3

  • 1Institute of Cardiovascular Disease, Key Lab for Arteriosclerology of Hunan province, Hengyang Medical School, University of South China, Hengyang, Hunan 421001, PR China; Department of Pathology, The First People's Hospital of Zigong, Zigong 643099, PR China.

The international journal of biochemistry & cell biology
|December 1, 2024
PubMed
概括

脂肪组织巨细胞 (ATM) 释放含有miR-500a-5p的外体,通过激活NLRP3炎症酶和降低脂肪细胞中的Nrf2调节,促进2型糖尿病 (T2DM) 中的炎症.

关键词:
脂肪细胞脂肪细胞外基因组是外基因组中的一个.巨细胞是一个巨细胞.在米R-500a-5p.在NLRP3炎症酶体中,NLRP3炎症Nrf2 没有任何问题.

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