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Updated: Jun 6, 2025

An Oncogenic Hepatocyte-Induced Orthotopic Mouse Model of Hepatocellular Cancer Arising in the Setting of Hepatic Inflammation and Fibrosis
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阻断CXCR3B的表达增加了瘤在肝细胞癌的侵略性

Hwan Hee Lee1,2, Seoyeon Oh1,2, Hyojeung Kang3

  • 1College of Pharmacy, Duksung Women's University, Seoul, Republic of Korea.

Anticancer research
|December 3, 2024
PubMed
概括

在肝细胞癌细胞中阻断CXCR3B增强了它们的迁移和入侵. 这也降低了天然杀手细胞对癌细胞的有效性,这表明CXCR3B抑制HCC.

关键词:
在CXCR3B中,CXCR3B是CXCR3B.在HCC中,HCC是HCC.在 NK 细胞中,NK 细胞是 NK 细胞.在SK-Hep1中使用.

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The orderly progression of the cell cycle depends on the activation of Cdk protein by binding to its cyclin partner. However, the cell cycle must be restricted when undergoing abnormal changes. Most cancers correlate to the deregulated cell cycle, and since Cdks are a central component of the cell cycle, Cdk inhibitors are extensively studied to develop anticancer agents. For instance, cyclin D associates with several Cdks, such as Cdk 4/6, to form an active complex. The cyclin D-Cdk4/6 complex...
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