在COVID-19感染后发生的气管支气管 Amyloidosis
Chathurangi Angammana1, Sameera Gamlath2, Ravini Karunathilake2
1University Medical Unit Colombo South Teaching Hospital Dehiwala-Mount Lavinia Sri Lanka.
Respirology case reports
|December 4, 2024
概括
在一名COVID-19后患有慢性咳的患者身上诊断出了一种罕见的气管支气管 Amyloidosis (TBA). 这突显了病毒感染可能引发罕见的炎症性肺病,如TBA.
科学领域:
- 肺部医学 肺部医学
- 病理学 病理学 病理学
- 免疫学 免疫学 免疫学
背景情况:
- 细胞外空间中的粉样蛋白沉积可能是不受控制的炎症的结果.
- 气管支气管粉样化症 (TBA) 是一种罕见的疾病,其特点是气道内粉样化物沉积物.
研究的目的:
- 在COVID-19后出现慢性咳的患者中报告一例气管支气管 Amyloidosis (TBA) 病例.
- 探索COVID-19和TBA发展之间的潜在病因联系.
主要方法:
- 一个68岁的女性在COVID-19后患有慢性咳的临床病例介绍.
- 诊断调查以确定持续呼吸道症状的原因.
主要成果:
- 诊断出气管支气管粉样性粉症 (TBA),涉及空气道中的粉样性沉积物.
- 与COVID-19的时间关联表明病毒诱导的炎症或免疫反应可能起作用.
结论:
- 这一案例强调了在COVID-19后患有持续呼吸道症状的患者中考虑罕见病因,如TBA的重要性.
- 进一步的研究可能是有必要的,以调查病毒感染和氨基粉症之间的潜在联系.
相关概念视频
COPD: Pathogenesis and Clinical Features
232
Chronic obstructive pulmonary disease (COPD) is a group of lung conditions that progressively worsen over time, including chronic bronchitis and emphysema. This cluster of diseases collectively leads to a gradual and irreversible decline in lung function over time.
The primary cause for the onset of COPD is cigarette smoking and exposure to air pollution. These hazardous factors initiate a chain reaction within the lungs, resulting in chronic inflammation, damage to the airways, and a...
The primary cause for the onset of COPD is cigarette smoking and exposure to air pollution. These hazardous factors initiate a chain reaction within the lungs, resulting in chronic inflammation, damage to the airways, and a...
232
Chronic Obstructive Pulmonary Disease-II: Pathophysiology
2.7K
Chronic Obstructive Pulmonary Disease (COPD) pathophysiology is intricate and multifaceted, involving a complex interplay of physiological processes. Understanding these mechanisms is crucial for effectively managing and treating COPD. Here is an in-depth look at the critical elements in the pathophysiology of COPD:
Chronic Inflammation
Chronic Inflammation
2.7K
COPD: Management Using Bronchodilators and Corticosteroids
178
Chronic obstructive pulmonary isease (COPD) involves a group of progressive lung disorders characterized by persistent airflow limitation and chronic respiratory symptoms. Asthma-COPD Overlap Syndrome (ACOS), encompassing features of both asthma and Chronic obstructive pulmonary disease (COPD), is a group of progressive lung disorders that includes chronic bronchitis, emphysema, and refractory (non-reversible) asthma. ACOS leads to complex clinical presentations that combine the inflammatory...
178
Chronic Obstructive Pulmonary Disease
1.2K
COPD is defined as a heterogeneous lung condition marked by persistent respiratory symptoms such as dyspnea, cough, and sputum production, caused by abnormalities in the airways that cause airflow obstruction.
Smoking is a primary risk factor for COPD, with over 80% of patients having a history of it. Patients typically experience progressive dyspnea or labored breathing, frequent coughing, and recurrent pulmonary infections. Many eventually succumb to respiratory failure, characterized by...
Smoking is a primary risk factor for COPD, with over 80% of patients having a history of it. Patients typically experience progressive dyspnea or labored breathing, frequent coughing, and recurrent pulmonary infections. Many eventually succumb to respiratory failure, characterized by...
1.2K
Asthma-II: Pathophysiology and Classification
2.6K
Asthma is a prevalent chronic respiratory condition marked by inflammation and hyperresponsiveness of the airways. Its pathophysiology involves complex interactions among inflammatory pathways, immune responses, and neural mechanisms.
Additionally, environmental and genetic factors play crucial roles in determining an individual's susceptibility to asthma and the severity of their condition.
Critical processes in asthma pathophysiology include:
Additionally, environmental and genetic factors play crucial roles in determining an individual's susceptibility to asthma and the severity of their condition.
Critical processes in asthma pathophysiology include:
2.6K
Pneumonia II: Pathophysiology
216
The pathophysiology of pneumonia involves the following steps:
216


