对石英的短期和长期病理反应是由在晶体破裂过程中形成的几乎自由的醇诱导的

Cristina Pavan1,2,3, Riccardo Leinardi4, Anissa Benhida4

  • 1Louvain Centre for Toxicology and Applied Pharmacology (LTAP), Institute of Experimental and Clinical Research (IREC), Université catholique de Louvain (UCLouvain), Brussels, Belgium. cristina.pavan@unito.it.

PubMed
概括

在破碎石英颗粒上的近自由 (NFS) 驱动肺炎,纤维化,癌症和自身免疫. 低NFS石英不会引起这些不良健康影响,这突显了NFS在毒性中的关键作用.

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