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乳酸细菌通过其生物结合能力和肠道环境修复能力来减少聚乙烯微型和纳米塑料诱导的毒性

Liuting Shi¹, Xin Teng², Changyin Wu¹

¹State Key Laboratory of Food Science and Resources, Jiangnan University, Wuxi, Jiangsu, 214122, PR China; School of Food Science and Technology, Jiangnan University, Wuxi, Jiangsu, 214122, PR China.

Environmental pollution (Barking, Essex : 1987)

|December 5, 2024

概括

乳酸细菌 (LAB) 可以减少微塑料和纳米塑料 (MNP) 暴露的毒性. 高结合性LAB菌株是有效的,肠道修复机制也有助于减轻MNP危害.

科学领域:

环境科学环境科学
微生物学微生物学
毒理学毒理学毒理学

背景情况:

微塑料和纳米塑料 (MNP) 是普遍存在的环境污染物.
关于减轻MNP毒性的数据有限,特别是在哺乳动物中.
乳酸细菌 (LAB) 是安全的食品级微生物,有可能与有害物质结合.

研究的目的:

研究乳酸细菌 (LAB) 在减少MNP诱导的小鼠毒性方面的疗效.
探索LAB体外MNP结合能力及其保护作用之间的关系.
了解LAB减轻MNP毒性的机制.

主要方法:

选择的LAB菌株具有不同的MNP结合能力,用于干预暴露于MNP的小鼠.
评估了LAB干预后MNP毒性降低的情况.
分析了紧结蛋白和肠道微生物群调节的作用.

主要成果:

具有更高的实验室MNP间隔能力的LAB菌株在减少MNP毒性方面表现出更大的有效性.
植物乳杆菌DT22,尽管低MNP结合,显著上调紧接口蛋白和调节肠道微生物群.
实验室对MNP毒性的缓解包括生物结合和肠道环境的修复.

结论:

LAB菌株显示出作为一种饮食干预措施的潜力,以抵消MNP诱导的毒性.
LAB的肠道修复能力是减轻MNP损害的关键因素,超出了简单的生物结合.
对基于LAB的环境污染物减轻策略进行进一步研究是有必要的.

关键词:

生物结合生物结合我们的肠道微生物群.乳酸细菌是一种乳酸细菌.微塑料和纳米塑料的使用

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