硫降低了与防御相关的WRKY转录因子的调节,并减弱了病原体相关的分子模式触发的免疫力
Dian Liu1, Joanna Jelenska2, Jessica M Morgan3
1Biochemistry and Molecular Biophysics, The University of Chicago, Chicago, Illinois, 60637, USA.
The Plant journal : for cell and molecular biology
|December 11, 2024
概括
植物硫蛋白 (PSK) 激素主要降低植物防御基因的调节,影响生长. WRKY转录因子是这种植物生长防御权衡的关键调节者.
科学领域:
- 植物分子生物学 植物分子生物学
- 植物激素信号传递器
- 植物与微生物的相互作用
背景情况:
- 硫 (PSK) 是一种促进植物生长的激素.
- 通过PSK受体 (PSKR1/PSKR2) 调解其作用.
- 似乎PSK信号抑制了植物的防御能力.
研究的目的:
- 调查PSK信号传递机制.
- 由PSK引起的个人资料转录变化.
- 监测PSK对工厂防御读数的影响.
主要方法:
- 用PSK治疗的阿拉比多普西斯的综合转录概况.
- 监测植物防御读数 (例如,MPK酸化,质沉积).
主要成果:
- PSK主要降低与防御相关的基因的调节,与与生长相关的基因的适度诱导.
- WRKY转录因子 (TFs) 是PSK响应基因的关键调节者.
- PSK治疗减少了PAMP反应,包括MPK酸化和质沉积.
结论:
- PSK信号编排了增长与防御的权衡,主要是通过抑制防御途径来实现.
- WRKY TFs对于PSK的监管作用至关重要,并且可能参与共享增长防御机制.
- 这些发现为植物激素交叉和免疫系统调节提供了洞察力.
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