昏迷状态和确定的形活动是由Amanita pantherina中毒引起的
Marco Cirronis1, Andrea Giampreti1, Georgios Eleftheriou1
1Bergamo Poison Center, ASST Papa Giovanni XXIII Hospital, Bergamo, Italy.
Toxicon : official journal of the International Society on Toxinology
|December 11, 2024
概括
阿曼尼塔 (Amanita pantherina) 类中毒是罕见的,通常会导致严重的神经症状,如昏迷和发作,原因是肌肉醇含量高. 迅速消化道去污染和 levetiracetam 治疗导致完全康复,没有持久的影响.
科学领域:
- 毒理学 毒理学 毒理学
- 神经学 神经学
- 菌类学 菌类学是指菌类学.
背景情况:
- 阿曼尼塔 (Amanita pantherina) 的中毒很少被记录,症状经常与阿曼尼塔 (Amanita muscaria) 重叠.
- A. pantherina含有更高度的肌肉醇,一种强大的神经毒素,增加了严重神经结果的风险.
- 准确识别有毒对于及时和适当的医疗干预至关重要.
更多相关视频
08:47Microdialysis of Excitatory Amino Acids During EEG Recordings in Freely Moving Rats
Published on: November 8, 2018
11.3K
06:28Author Spotlight: Unraveling Seizure Dynamics and Novel Therapeutics for Status Epilepticus Using CMOS High-Density Microelectrode Array Systems
Published on: September 27, 2024
2.2K
相关概念视频
Physical Properties of Amines
3.2K
Amines with low molecular weight are usually gaseous at room temperature, while those with high molecular weight are liquid or solids in nature. Usually, low molecular weight amines have a rotten fish-like smell. Diamines typically have a pungent smell. For instance, cadaverine and putrescine, depicted in Figure 1, are two molecules responsible for decaying tissue.
3.2K
Anticholinesterase Agents: Poisoning and Treatment
2.0K
Anticholinesterases, also known as cholinesterase inhibitors, work by blocking the breakdown of acetylcholine, leading to its accumulation in the synaptic cleft. This accumulation indirectly enhances both muscarinic and nicotinic actions. These agents are classified as reversible or irreversible based on their mechanism of action.
Irreversible agents form a strong bond with the cholinesterase enzyme, making it inactive. The breakdown of the phosphorylated enzyme is...
Irreversible agents form a strong bond with the cholinesterase enzyme, making it inactive. The breakdown of the phosphorylated enzyme is...
2.0K
Hepatic Encephalopathy
53
DefinitionHepatic encephalopathy is a reversible neurologic syndrome that results from advanced liver dysfunction or portosystemic shunting. It leads to disturbances in cognition, behavior, and motor function due to the brain’s exposure to gut-derived toxins that the liver fails to detoxify.EtiologyThis condition develops either in the setting of acute fulminant hepatitis or progressively during chronic liver disease, such as cirrhosis and portal hypertension. Portosystemic...
53
