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乌罗立A和尼古丁胺 рибоoside在人类微质细胞中差异调节先天免疫防御和新陈代谢

Helena Borland Madsen1, Claudia Navarro1, Emilie Gasparini1

  • 1Center for Healthy Aging, Department of Cellular and Molecular Medicine, University of Copenhagen, Copenhagen, Denmark.

Frontiers in aging neuroscience
|December 12, 2024
PubMed
概括

乌罗立A (UA) 和尼古丁胺里化物 (NR) 显示出与衰老相关的细胞衰退的潜在作用. 这两种化合物都减少衰老,并影响神经炎症和线粒体功能,这表明神经退行性疾病的治疗效益.

关键词:
老化的老化 衰老的老化天生的免疫信号传递.微质细胞中的微质细胞线粒体健康 线粒体健康尼古丁胺胺里化物乌罗利A是一种尿素.

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