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Updated: Jun 27, 2026

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西诺因调节通过CHRNA7诱导的体质过敏引起的代谢重编程

Jie Zhao1, Zi Wang1, Xinxin Zou1

  • 1State Key Laboratory of Natural Medicines, School of life science and technology, China Pharmaceutical University, Nanjing 211000, PR China.

Life sciences
|December 26, 2024
PubMed
概括

西诺因 (SIN) 通过改变代谢重编程,特别是葡萄糖和谷氨酸代谢,影响败血症. 这通过与尼古丁乙胆受体α7亚单元 (CHRNA7) 的相互作用发生,为败血症治疗提供一种代谢方法.

关键词:
这就是CHRNA7的原因.代谢重编程是一种代谢重编程.败血症 这是一种败血症.这就是"sinomenine".

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