电动肌肉刺激诱导的肌肉收缩对内源性疼痛调节系统的影响:定量感官测试评估
Satoshi Ohga1, Takafumi Hattori2, Kazuhiro Shimo2,3
1Faculty of Rehabilitation, Kobe Gakuin University, 518 Arise, Ikawadani-cho, Nishi-ku, Kobe, Hyogo, 651-2180, Japan. ohga@reha.kobegakuin.ac.jp.
BMC musculoskeletal disorders
|December 28, 2024
概括
应用到四头肌的电动肌肉刺激 (EMS) 在局部增加了疼痛耐受性. 这表明EMS的缓解疼痛效果局限于刺激的肌肉,而不是全身.
科学领域:
- 疼痛感知和调制的疼痛感知和调制.
- 运动生理学 运动生理学
- 神经肌肉电刺激是一种神经肌肉电刺激.
背景情况:
- 运动诱导的低痛症 (EIH) 减少了运动后的疼痛感知,由中央和外围机制调解.
- 在EIH中孤立肌肉收缩的特定作用尚未完全理解.
- 之前关于电动肌肉刺激 (EMS) 的研究集中在局部疼痛缓解上,通常使用主观措施.
研究的目的:
- 调查EMS诱导的肌肉收缩对系统性止痛的贡献,独立于运动皮层活动.
- 通过分析骨肌肉质量 (SMM),骨肌肉质量指数 (SMI) 和条件疼痛调制 (CPM) 来探索EIH的机制.
主要方法:
- 一项涉及27名健康成年人的交叉研究,比较EMS和假冒干预.
- 测量包括骨肌量 (SMM),骨肌量指数 (SMI),条件疼痛调制 (CPM) 和压力疼痛值 (PPT).
- 在没有关节运动的情况下,EMS应用于四头四骨20分钟.
主要成果:
- 压力疼痛值 (PPT) 在EMS后刺激的四头肌显著增加,但在非收缩部位并非如此.
- 在PPT和SMM和SMI的变化之间观察到强烈的正相关性.
- 在假条件或CPM中没有发现显著的影响.
结论:
- 由EMS诱导的肌肉收缩的镇痛作用似乎局限于刺激的肌肉.
- 在这种情况下,中央疼痛调节机制似乎不是EMS诱导的镇痛的主要驱动因素.
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