向S100A9通过调节自闭症小鼠模型中的神经炎症和髓化来减轻社会功能障碍

Hong Gong1, Yao Lu2, Shi-Long Deng3

  • 1Department of Military Cognitive Psychology, School of Psychology, Third Military Medical University (Army Medical University), Chongqing 40038, China.

Pharmacological research
|December 29, 2024
PubMed
概括

微质衍生的S100A9在自闭症模型中驱动神经炎症和社会缺陷. 抑制S100A9在治疗自闭症相关的神经发育障碍方面表现有前途.