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Updated: May 7, 2025

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托马提丁通过激活Nrf2/HO-1/GPX4信号通路来缓解椎间盘退化

Ze Li1,2, Pu Cheng1,2, Huifeng Xi1,2

  • 1Department of Orthopaedics, Taizhou Hospital Affiliated to Wenzhou Medical University, Linhai, Zhejiang Province, People's Republic of China.

Drug design, development and therapy
|January 1, 2025
PubMed
概括

托马提丁通过激活Nrf2/HO-1/GPX4通路来缓解椎间盘退化 (IDD),从而减少细胞核中的铁亡. 这表明tomatidine是IDD的有希望的治疗候选者.

关键词:
在GPX4中使用GPX4.第2章 没有了铁性化 (ferroptosis) 是一种椎间盘退化 椎间盘退化番茄胺是什么意思

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The transcription factor NF-κB was discovered in 1986 in the lab of Nobel laureate Professor David Baltimore, for its interaction with the immunoglobulin light chain enhancer in B-cells. After more than three decades of study, it is now evident that NF-κB regulates the expression of over 100 genes. Most of these genes play an essential role in the innate and adaptive immune responses as well as the inflammatory responses of animals.
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