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[耐火性患者的微量营养素摄入量与性饮食治疗]
Silvia Velandia1, Patricio Astudillo2, Keryma Acevedo2
1Facultad de Medicina, Departamento de Gastroenterología y Nutrición Pediátrica, Pontificia Universidad Católica de Chile, Santiago, Chile.
Andes pediatrica : revista Chilena de pediatria
|January 6, 2025
概括
性饮食 (KD) 可以导致微量营养素缺乏,特别是经典形式. 经过修改的阿特金斯饮食 (MAD) 配有特殊配方,为治疗耐药性和代谢障碍提供更充分的营养摄入量.
科学领域:
- 营养科学 营养科学
- 儿科神经学 儿科神经学
- 代谢障碍 代谢障碍 代谢障碍
背景情况:
- 素饮食 (KD) 是一种重要的非药物治疗药物耐药性 (DRE) 和像Glut-1缺乏症这样的代谢天生的错误.
- KD的限制性往往导致微量营养素缺乏,在不同的KD模式中存在差异.
- 了解这些变异对于优化患者护理至关重要.
研究的目的:
- 评估和比较各种类型的饮食中微量营养素的摄入量.
- 在不同的KD方法中识别存在缺乏风险的特定微量营养素.
主要方法:
- 一项观察性,横截面研究评估了19名患有DRE或Glut-1缺乏症的儿科患者.
- 对21种微量营养素的饮食摄入量进行了分析,基于KD类型 (经典KD,修改的阿特金斯饮食[MAD]),配方使用和适应推的饮食摄入量 (RDI).
- 缺陷被定义为<75%的RDI.
主要成果:
- 微量营养素缺乏是普遍存在的,影响了76.2%的古典KD和42.9%的MAD.
- 与MAD相比,古典KD的维生素D,B2,B12,,,和摄入量明显较低.
- 虽然一些维生素摄入量超过了RDI的100%,但注意到了特定的缺陷,配方改善了MAD的充足性.
结论:
- 大多数饮食模式导致微量营养素摄入不足.
- 经过修改的阿特金斯饮食 (MAD) 与特殊配方相结合,证明了最充分的微量营养素配置.
- 营养监测和有针对性的补充对于接受KD治疗的患者至关重要.
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