患有低度脑瘤引起的患者的同时出现的焦虑和抑郁症
Sungel Xie1, Jiao Qiao2, Guoming Luan2
1Department of Neurosurgery, Affiliated Hospital of Zunyi Medical University, Dalian Road 149, Huichuan District, Zunyi, 563000, Guizhou Province, China. xiemg1991@163.com.
Scientific reports
|January 9, 2025
概括
焦虑和抑郁经常影响由于低度脑瘤 (LBTs) 引起的患者. 早期查和干预至关重要,特别是对于那些有不一致的EEG发现或瘤位置的人来说.
科学领域:
- 神经学 神经学
- 精神病学是一个精神病学.
- 神经瘤学神经瘤学
背景情况:
- 是一种常见的神经系统疾病.
- 低度脑瘤 (LBTs) 可能导致.
- 同时出现的精神疾病,如焦虑和抑郁症,在患者中越来越多地被发现.
研究的目的:
- 为了评估患有LBT相关的患者焦虑和抑郁的患病率.
- 为了确定与这些精神病相关的风险因素.
主要方法:
- 来自107名接受LBT手术的患者的临床数据的回顾性审查.
- 在手术前对焦虑和抑郁症进行神经心理评估.
- 单变量和多变量分析以确定风险因素.
主要成果:
- 19.6%的患者患有焦虑症,24.3%患有抑郁症.
- 不一致的内脉脑电图 (EEG) 发现与焦虑有关 (OR=3.35).
- 不一致的阴道EEG发现 (OR=4.44) 和瘤位置 (OR=13.52) 与抑郁症有关.
结论:
- 焦虑和抑郁是LBTs继发性的常见并发症.
- 早期查和心理健康干预是必不可少的.
- 患有不和性性泄漏或时间瘤参与的患者需要特别注意.
相关概念视频
Electroconvulsive Therapy
22
Electroconvulsive therapy (ECT), or shock therapy, remains a critical biomedical intervention for severe, treatment-resistant depression. While its origins can be traced back to Hippocrates' observations that malaria-induced convulsions alleviated mental illness, modern ECT has evolved significantly from its earlier, more primitive applications. First introduced in 1938 by Ugo Cerletti and his colleagues, ECT involves inducing controlled seizures using electrical currents. In its early...
22
Arteries of the Lower Limbs
177
Epilepsy is a chronic neurological disease marked by recurrent, unpredictable seizures. These seizures are caused by abnormal electrical discharges in the brain, leading to behavior, sensation, or consciousness alterations. They can also cause transient impairment of awareness, interfering with daily activities.
Various factors can trigger epilepsy, including genetic factors, brain damage, metabolic causes, and unknown etiology. Diagnosis of epilepsy involves electroencephalography (EEG), which...
Various factors can trigger epilepsy, including genetic factors, brain damage, metabolic causes, and unknown etiology. Diagnosis of epilepsy involves electroencephalography (EEG), which...
177
Depression: Overview
213
Depression is a prevalent mental illness marked by persistent sadness and lack of interest in previously enjoyable activities. It can take several forms, including major depression, persistent depressive disorder, and bipolar I and II disorders. Symptoms range from emotional changes like chronic worry to physical changes like sleep disturbances and suicidal thoughts. From a neurobiological perspective, depression is believed to be triggered by abnormalities in the brain's prefrontal cortex,...
213
Generalized Anxiety Disorder
45
Generalized Anxiety Disorder (GAD) is a chronic condition characterized by excessive and uncontrollable worry that persists for at least six months, significantly interfering with daily functioning. Unlike situational anxiety, which arises in response to specific stressors, GAD often occurs without a clear cause. Individuals may experience disproportionate worry about work, health, or relationships. For instance, a person might continuously fear poor health despite normal medical evaluations or...
45
Seizures: Classification
300
Epilepsy is primarily characterized by unpredictable seizures, either provoked by an identifiable factor, such as injury or illness, or unprovoked, occurring spontaneously without apparent cause.
Seizures are typically classified into two main categories: focal and generalized seizures.
Focal Seizures
Focal seizures originate from specific regions of the brain. These seizures are further sub-classified into two types:
Seizures are typically classified into two main categories: focal and generalized seizures.
Focal Seizures
Focal seizures originate from specific regions of the brain. These seizures are further sub-classified into two types:
300
Anxiolytic Drugs: Benzodiazepines and Buspirone
499
Benzodiazepines are a class of anxiolytic drugs known for their rapid efficacy and high therapeutic-to-lethal dose ratio, but with a potential risk of drug dependence. These drugs are lipophilic, allowing for rapid absorption after oral administration, eventually reaching the central nervous system (CNS). Once in the CNS, benzodiazepines bind to the allosteric site of the GABAA receptor. This binding enhances the inhibitory effects of the neurotransmitter GABA. By doing so, they prevent...
499


