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Updated: May 30, 2025

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通过CB1R介导的施万细胞中不适应的外周生成有助于糖尿病神经病变

Weizhen Li1,2, Tuo Yang1,2, Ningning Wang1,2

  • 1Department of Hand and Foot Surgery, China-Japan Union Hospital of Jilin University, Changchun, 130033, China.

Advanced science (Weinheim, Baden-Wurttemberg, Germany)
|January 31, 2025
PubMed
概括

糖尿病外围神经病变涉及斯万细胞中的异常脂肪代谢,由CB1R-Hmgcs2通路驱动. 阻断这种通路可以改善神经功能,并为糖尿病神经损伤提供新的治疗点.

关键词:
大麻素类型1受体受体糖尿病周围神经病变ketogenesis 是一种基因的形成.代谢重编程是指代谢重编程.施万的细胞细胞.

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