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Updated: May 29, 2025

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坦辛拉克通过NRF2激活引发乳腺癌细胞中的甲状腺分裂

Wanjun Lin1, Zifeng Huang1, Xuening Zhang1

  • 1State Key Laboratory of Quality Research in Chinese Medicine, Macau University of Science and Technology, Macau, China.

Frontiers in pharmacology
|February 5, 2025
PubMed
概括

坦辛拉克通过NRF2激活诱导灾难性巨细胞 (甲) 形成ER+和HER2+/EGFR+乳腺癌,从而选择性向ER+和HER2+/EGFR+乳腺癌. 这种化合物在治疗耐药乳腺癌方面表现有前途,副作用可能更少.

关键词:
在NRF2激活过程中,乳腺癌 乳腺癌 乳腺癌灾难性的巨型皮诺细胞形成.药物耐药性 耐药性 药物耐药性甲症是指甲症的发生.坦辛酸氨的使用方法

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