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Updated: May 27, 2025

Quantitative 3D In Silico Modeling q3DISM of Cerebral Amyloid-beta Phagocytosis in Rodent Models of Alzheimer's Disease
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揭示了KLF9-介导的IFITM3调节在粉样蛋白生成中的作用

Yijia Feng1,2,3, Qian Zhou4, Bolang Hu1,2,3

  • 1Center for Geriatric Medicine, The First Affiliated Hospital, Wenzhou Medical University, Wenzhou, Zhejiang, China.

FASEB journal : official publication of the Federation of American Societies for Experimental Biology
|February 15, 2025
PubMed
概括

克鲁佩尔类因子9 (KLF9) 调高了干扰素诱导的跨膜蛋白3 (IFITM3) 表达,增加了阿尔茨海默氏症患者的粉样β (Aβ) 生产.

关键词:
阿尔茨海默病的疾病阿尔茨海默病的疾病.亚博体育在线娱乐 亚博体育在线娱乐如果IFITM3是IFITM3,那么它是什么?在KLF9KLF9KLF9转录规则 转录规则 转录规则

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