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Updated: May 27, 2025

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衰老的内皮细胞衍生的加列3通过内皮纤维细胞和内皮巨细胞交叉促进了化

Demin Cheng1, Wenxiu Lian2, Xinying Jia2

  • 1Department of Occupational Medicine and Environmental Toxicology, Nantong Key Laboratory of Environmental Toxicology, School of Public Health, Nantong University, Nantong 226019, China; Department of Occupational Medical and Environmental Health, Key Laboratory of Modern Toxicology of Ministry of Education, Center for Global Health, School of Public Health, Nanjing Medical University, Nanjing 211166, China.

Journal of hazardous materials
|February 16, 2025
PubMed
概括

菌性肺纤维化由衰老的内皮细胞分泌加勒3 (Gal3) 恶化. Gal3促进纤维细胞和巨细胞的激活,推动疾病的进展. 向Gal3可以治疗这种不可逆转的肺部疾病.

关键词:
在CEBPBP的基础上,CEBPBP加尔3加尔3加尔3加尔3衰老的衰老 衰老的衰老细胞内皮细胞的内皮细胞.在化中,有化.

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