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Updated: May 5, 2026

Preparation of Acute Hippocampal Slices from Rats and Transgenic Mice for the Study of Synaptic Alterations during Aging and Amyloid Pathology
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产前高homocysteinemia导致突触功能障碍和结构变化在CA1大鼠海马的老鼠

Tatyana Y Postnikova1, Alexandra V Griflyuk1, Natalia L Tumanova1

  • 1Sechenov Institute of Evolutionary Physiology and Biochemistry, Russian Academy of Sciences, 194223 St. Petersburg, Russia.

Biomolecules
|February 26, 2025
PubMed
概括

产前暴露在高水平的同类氨酸 (HCY) 中会损害海马突触功能,并导致成年大鼠的结构变化. 这些缺陷仍然存在,这表明与神经发育障碍有联系.

关键词:
CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1 CA1激发性神经传递的激发性神经传递现场 postsynaptic 潜力 在这个领域的 postsynaptic 潜力在海马体内,海马体同型半氨酸同型半氨酸最大的电击值测试试验产前发育 产前发育突触囊泡中的突触囊泡.

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