Prdx5通过调节TLR4/NF-κB通路来调节巨细胞极化,以促进慢性前列腺炎中亡

Weikang Wu1, Tong Meng1, Yufan Wang1

  • 1Department of Urology, The First Affiliated Hospital of Anhui Medical University, Institute of Urology, and Anhui Province Key Laboratory of Urological and Andrological Diseases Research and Medical Transformation, Anhui Medical University, Hefei 230022, Anhui, China.

PubMed
概括

百氧化素5 (Prdx5) 促进慢性前列腺炎/慢性盆腔疼痛综合征 (CP/CPPS) 通过增加M1巨细胞两极分化和前列腺细胞亡. 准Prdx5可能为CP/CPPS提供新的治疗策略.

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