在患有高血压和2型糖尿病的患者中,足度检查结果的关联:回顾性分析
Christopher Paschen1, Maximilian C Koeller2, Helga Schachner2
1Division of Nephrology and Dialysis, Department of Medicine III (C.P., K.D., M.K., M.H., R.O.), Medical University of Vienna, Austria.
动脉高血压和2型糖尿病会导致脏受损,导致细胞损失. 然而,在高血压患者中,2型糖尿病不会使这些脏变化恶化.
科学领域:
- 腎臟病學 (nephrology) 是一種醫學專業.
- 病理学 病理学 病理学
- 糖尿病学 糖尿病学
背景情况:
- 动脉高血压 (HTN) 和2型糖尿病 (T2DM) 是慢性病 (CKD) 进展的关键因素.
- 淋巴细胞膨胀和细胞损失是与HTN和T2DM相关的CKD的特征特征.
- 了解HTN的形态变化和T2DM的影响对于治疗病至关重要.
研究的目的:
- 为了研究患有动脉高血压的患者的质细胞和细胞的形态变化.
- 确定并存的2型糖尿病对高血压患者这些变化的贡献.
- 为了评估形态参数和脏切除术后功能补偿之间的关联.
主要方法:
- 99名患者瘤切除术脏组织的回顾式立体分析.
- 在HTN,HTN+T2DM和对照组之间比较形态特征 (球粒体积,细胞密度,细胞核体积).
- 线性回归模型用于评估形态参数对估计的膜过率 (eGFR) 影响的线性回归模型在腎切除术后发生变化.
主要成果:
- 与对照组相比,HTN+T2DM患者的足细胞密度降低了 (P=0.02).
- 细胞核体积在HTN+T2DM患者和对照组中较大 (P=0.004).
- 淋巴细胞体积,降低细胞密度和核缩与脏补偿受损相关 (P<0.01).
结论:
- 在HTN和HTN+T2DM中,细胞枯竭和核缩独立地发生.
- 同时存在的T2DM似乎不会在高血压患者中加剧淋巴细胞或细胞的变化.
- 形态变化与脏手术后较差的功能恢复有关.
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