[毒素A和牛氨基酶氧化剂的连续使用,用于纠正中风后的性]
D A Krasavina1, D O Ivanov1, O R Orlova2
1Saint Petersburg State Pediatric Medical University, Saint-Petersburg, Russia.
Zhurnal nevrologii i psikhiatrii imeni S.S. Korsakova
|March 6, 2025
概括
这项案例研究表明,A型肉毒素和阿佐克西默牛氨基酶有效治疗缺血性中风后的性. 联合治疗改善了69岁患者的肌肉弹性和运动功能.
科学领域:
- 神经学 神经学
- 康复医学 康复医学 康复医学
- 临床案例研究临床案例研究
背景情况:
- 缺血性中风可能导致半衰和性,影响运动功能.
- 和收缩形成在中风后的康复过程中存在重大挑战.
- 毒素A型 (BTA) 是一种被公认的治疗肌肉的方法.
研究的目的:
- 为了呈现一种临床案例,结合毒素A型 (BTA) 和阿佐克西默牛氨基酶 (BA) 治疗后性缺血性中风性.
- 评估这种联合治疗方法的安全性,耐受性和有效性.
- 评估对肌肉度,组织结构和运动功能的影响.
主要方法:
- 一个69岁的病人与右侧半衰减后缺血性中风治疗.
- 毒素A型 (incobotulinumtoxinA) 在超声波指导下进行.
- 在BTA治疗后,阿佐克西默牛氨基酶被注射到纤维化肩膀肌肉中.
- 使用修改的阿什沃思尺度 (MAS),阴影测量和电肌图学来监测治疗效果.
主要成果:
- 毒素A型注射导致上肢性 (MAS尺度) 的减少.
- 随后的阿佐克西默牛氨基酶应用导致肌肉组织结构的改变和肌肉度的降低.
- 结合疗法表现出良好的耐受性和有效性,用于控制缩形成的性.
- 观察到肌肉弹性和运动功能的改善.
结论:
- 一贯使用甲型肉毒素和氧化物牛氨基酶对中风后性是安全有效的.
- 阿佐克西默牛氨基酶通过减少纤维化和改善肌肉弹性来增强肉类治疗的效果.
- 这种综合方法为改善病患中风后动和收缩的运动功能提供了有希望的策略.
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