基于FKBPL的治疗性,AD-01,通过稳定缺氧诱导因子-α和炎症,保护内皮免受缺氧诱导的损伤

Sahar Ghorbanpour1,2, Siân Peta Cartland2, Hao Chen1

  • 1School of Life Sciences & Institute for Biomedical Materials and Devices, Faculty of Science, University of Technology Sydney, Sydney, NSW, Australia.

概括

像FKBPL这样的FK506结合蛋白在心血管疾病中起作用. AD-01在缺氧下恢复内皮功能,为心血管疾病中的内皮细胞功能障碍提供了潜在的治疗方法.

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