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Updated: May 22, 2025

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血小板激活刺激巨细胞通过PF4/CXCR3信号传递来增强性结肠炎

Yuxiao Niu1, Anhong Li2, Weihua Xu3

  • 1Graduate School, Xinxiang Medical University, Xinxiang, Henan 453003, P.R. China.

International journal of molecular medicine
|March 14, 2025
PubMed
概括

血小板通过通过血小板因子4 (PF4) /C-X-C动机化学因子受体3 (CXCR3) 途径激活巨细胞来加剧性结肠炎 (UC). 抑制这种途径为UC提供了潜在的新疗法.

关键词:
灭症 (apoptosis) 是一种死亡的过程.这是一种炎症炎症炎症炎症.巨细胞是一个巨细胞.血小板 血小板 血小板是什么?血小板因子4/C-X-C动机化学因子受体3 血小板因子4/C-X-C动机化学因子受体3性结肠炎是一种

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