相关实验视频

Updated: May 6, 2026

Oral Health Assessment by Lay Personnel for Older Adults

Oral Health Assessment by Lay Personnel for Older Adults

Published on: February 2, 2020

痴呆症患者的空白状况:系统性审查

Weigao Cheng¹, Dongmin Zhang^1,2, Qiwen Li¹

¹State Key Laboratory of Oral & Maxillofacial Reconstruction and Regeneration, Key Laboratory of Oral Biomedicine Ministry of Education, Hubei Key Laboratory of Stomatology, School & Hospital of Stomatology, Wuhan University, Wuhan 430072, China.

Journal of clinical medicine

|March 17, 2025

概括

痴呆症患者的口腔健康状况明显较差,与对照组相比,牙和牙缺失率更高. 这凸显了迫切需要加强对痴呆症患者的牙科护理的关注.

科学领域:

老年学是一门学科.
口腔健康口腔健康
神经学神经学

背景情况:

痴呆症患者经常经历口腔健康受损.
现有的关于痴呆症患者病状况的研究提出了相互矛盾的发现.
这项研究解决了对这一群体口腔健康的全面评估的需要.

研究的目的:

在被诊断患有痴呆症的老年人中系统地审查和评估状况.
量化患有痴呆症和没有痴呆症的个体之间的口腔健康指标差异.

主要方法:

在PubMed,Web of Science,Embase和Scopus数据库中系统地搜索文献.
使用纽卡斯尔-太华尺度和乔安娜·布里格斯研究所批判性评估清单进行偏见风险评估.
使用随机效应模型对腐烂,缺失,填充牙 (DMFT) 指数及其组件 (DT,MT,FT) 的元分析.

主要成果:

对20项研究的元分析显示,痴呆症组的DMFT指数 (WMD:3.76) 在痴呆症组显著更高.
患有痴呆症的个体表现出牙腐烂 (DT) 的增加 (WMD:0.40) 和牙缺失 (MT) (WMD:3.67).
两组之间填充牙 (FT) (WMD: -0.66) 没有显著差异.

结论:

与不痴呆的人相比,患有痴呆症的人表现出较差的状况.
这些发现强调了加强口腔健康监测和对痴呆症患者的护理的迫切需要.
医务人员和护理人员应优先考虑痴呆症患者的口腔卫生.

关键词:

这就是阿尔茨海默病的原因.皮肤腐烂,皮肤腐烂.痴呆症痴呆症是一种痴呆症.口腔健康是指口腔的健康状况.

更多相关视频

Evaluation of the Cognitive Performance of Hypertensive Patients with Silent Cerebrovascular Lesions

Evaluation of the Cognitive Performance of Hypertensive Patients with Silent Cerebrovascular Lesions

Published on: April 23, 2021

Dual-Task Stroop Paradigm for Detecting Cognitive Deficits in High-Functioning Stroke Patients

Dual-Task Stroop Paradigm for Detecting Cognitive Deficits in High-Functioning Stroke Patients

Published on: December 16, 2022

相关概念视频

Alzheimer's Disease: Overview

Alzheimer's Disease: Overview

Alzheimer's Disease (AD) is a continually advancing neurodegenerative disorder, distinguished by escalating memory loss, cognitive dysfunction, and dementia. The disease unfolds in three stages: preclinical, mild cognitive impairment (MCI), and dementia. Its onset is insidious, and the progression gradual, with the cause not well explained by other disorders.
The clinical diagnosis of AD hinges on the presence of memory and other cognitive impairments. Biomarkers, such as changes in Aβ...

Alzheimer's Disease: Treatment

Alzheimer's Disease: Treatment

Alzheimer's Disease (AD), a neurodegenerative disorder, is pathologically identified by amyloid plaques and neurofibrillary tangles composed of tau protein. AD pharmacotherapy aims to manage cognitive symptoms, delay disease progression, and treat behavioral symptoms. The treatment is primarily symptomatic and palliative, with no definitive disease-modifying therapy available. Cholinesterase inhibitors, including donepezil (Aricept), rivastigmine (Exelon), and galantamine (Razadyne), are...

Dementia

Dementia

Dementia is a collective term for cognitive disorders primarily affecting memory, thinking, and reasoning. It is not a specific disease but a syndrome, with Alzheimer's disease being the most common cause, accounting for approximately 60-80% of cases. Other types include vascular dementia, Lewy body dementia, and frontotemporal dementia. Dementia affects millions worldwide, particularly older adults, though it is not a normal part of aging.
The progression of dementia is generally gradual....

Alzheimer Disease l: Introduction

Alzheimer Disease l: Introduction

Alzheimer disease is a chronic, progressive, and irreversible neurodegenerative disorder and the most common cause of dementia in older adults. It leads to gradual neuronal loss, causing cognitive decline, behavioral changes, and loss of functional independence.Risk Factors and EtiologyThe disease is multifactorial. Age is the strongest risk factor, with prevalence doubling every 5 years after age 65. Genetic factors include mutations in genes such as APP, PSEN1, and PSEN2, which are associated...

Alzheimer Disease ll: Pathophysiology

Alzheimer Disease ll: Pathophysiology

Alzheimer disease involves structural changes in the brain that begin long before symptoms appear. The most distinctive features are extracellular neuritic plaques and intracellular neurofibrillary tangles.Neuritic plaques form in the cerebral cortex and around blood vessels. These plaques contain a dense core of beta-amyloid (Aβ)—a toxic protein fragment that clumps outside neurons. The core is surrounded by damaged neuronal extensions, as well as reactive astrocytes and...

Dementia l: Introduction

Dementia l: Introduction

Dementia is an acquired, progressive syndrome characterized by a decline in multiple cognitive domains severe enough to impair daily functioning and reduce independence. Although memory loss is a central feature, the diagnosis requires additional deficits involving language, executive function, visuospatial skills, judgment, calculation, or abstract reasoning. These cognitive impairments reflect underlying neurodegenerative or vascular processes that gradually disrupt neuronal networks...