缺少Atp6v0d2部分恢复了缺少Mcoln1的小鼠红色体的缺陷

Yuehuan Li1, Ahmed E El Zowalaty1,2, Jonathan Matthew Hancock1,2

  • 1Department of Physiology and Pharmacology, College of Veterinary Medicine, University of Georgia, Athens, GA 30602, USA.

概括

由于红体 (CL) 退化,Mcoln1缺乏导致小鼠的不孕症. 缺少ATP6V0d2部分挽救了Mcoln1淘汰赛小鼠的CL功能和生育能力,但并没有完全恢复孕激素的产生.