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Updated: Jun 21, 2026

Hyperinsulinemic-euglycemic Clamps in Conscious, Unrestrained Mice
11:10

Hyperinsulinemic-euglycemic Clamps in Conscious, Unrestrained Mice

Published on: November 16, 2011

MIR192 抑制了GLP-1受体,并改善了因他类药物引起的胰岛素分泌障碍

Yu-Lin Kuang1, Cassandra A A Locatelli2,3, Yuanyuan Qin1

  • 1Department of Pediatrics, University of California San Francisco, Oakland, CA, USA.

bioRxiv : the preprint server for biology
|April 1, 2025
PubMed
概括

类药物可以增加新发糖尿病的风险. 这项研究揭示了microRNA-192 (miR-192) 调节GLP-1受体,影响葡萄糖刺激的胰岛素分泌,并可能解释他类药物诱导的糖尿病风险.

关键词:
通过RNA测序进行RNA测序.电子健康记录 电子健康记录葡萄糖类1受体受体葡萄糖刺激的胰岛素分泌物诱导多能干细胞的诱导干细胞.miR-192是192年的一款飞机.新出现的糖尿病.胰腺β细胞是什么药物基因组学 药物基因组学斯塔丁是一个国家.

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