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Updated: May 13, 2025

Mechanism of Kemeng Fang's Inhibition of Podocyte Apoptosis in Rats with Membranous Nephropathy through the PI3K/AKT Signaling Pathway
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Published on: August 23, 2024

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聚达丁通过诱导亡并通过p38-MAPK通路激活自性来保护白血病

Wenhui Zhang1, Zhongwen Liu1, Peiyuan Hao2

  • 1Department of Hematology, Henan Provincial People's Hospital, Zhengzhou University People's Hospital, Zhengzhou, 450002, Henan, China.

Naunyn-Schmiedeberg's archives of pharmacology
|April 14, 2025
PubMed
概括

聚达丁,来自Polygonum cuspidatum,显示在治疗急性髓性白血病 (AML) 的承诺. 它通过p38-MAPK通路激活自,抑制癌细胞的增殖并诱导细胞亡.

关键词:
急性骨髓性白血病 (AML) 是一种急性骨髓性白血病.细胞灭亡 (apoptosis) 是一种死亡的过程.自自是一种自的过程.在P38-MAPKK中使用.聚达丁是一种多元素.

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