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Updated: May 10, 2025

Using the Activity-based Anorexia Rodent Model to Study the Neurobiological Basis of Anorexia Nervosa
07:46

Using the Activity-based Anorexia Rodent Model to Study the Neurobiological Basis of Anorexia Nervosa

Published on: October 22, 2015

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在基于活动的厌食症模型中,慢性饥饿诱导了微质细胞的枯竭

Valerie Verspohl1, Miranda van Egmond1, Lilly Kneisel1

  • 1Institute of Neuroanatomy, RWTH Aachen University, Wendlingweg 2, 52074, Aachen, Germany.

Scientific reports
|April 24, 2025
PubMed
概括

神经性厌食症 (AN) 导致大脑体积减少. 在老鼠中,慢性饥饿使质细胞枯竭,这表明这是大脑缩的基础. 欧米茄-3脂肪酸和益生菌可能有助于减少AN症状.

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During the initial hours of fasting, the body uses up its glycogen stores as an energy source. Once these glycogen reserves are depleted, the body begins breaking down stored triglycerides and structural proteins. During this stage, glycerol becomes a key substrate for gluconeogenesis, while free fatty acids undergo beta-oxidation to provide energy for tissues, such as skeletal muscle. In the fasting state, the body spares protein breakdown as much as possible to conserve muscle and structural...
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