通过促进PI3K介导的巨细胞M1极化,以碳点为基础治疗细菌性肺炎

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Xuan Jiang1, Jun Wang1, Lin Gan1

  • 1State Key Laboratory for Diagnosis and Treatment of Severe Zoonotic Infectious Diseases, Key Laboratory for Zoonosis Research of the Ministry of Education Institute of Zoonosis, College of Veterinary Medicine, Jilin University, Changchun, 130062, China.

Journal of nanobiotechnology
|April 26, 2025
PubMed
概括

新的碳点 (CDots) 增强了免疫细胞对抗耐药细菌的功能. 这些CDot向PIK3CD,促进巨细胞两极分化,改善小鼠的细菌性肺炎治疗.

关键词:
这是一种细菌性肺炎.这些碳点是碳点.巨细胞的两极分化具有多种药物耐药性的药物.这是一个PIK3CDCD.

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