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对PM_2.5暴露和流感风险增加之间的关联进行了系统审查和元分析

Ava Orr^1,2, Rebekah L Kendall¹, Zeina Jaffar¹

¹Center for Environmental Health Sciences, Biomedical & Pharmaceutical Sciences, University of Montana, Missoula, MT, United States.

Frontiers in epidemiology

|April 28, 2025

概括

暴露于细颗粒物 (PM2.5) 增加了流感风险,特别是在极端温度下. 本综述综合了有关空气污染与流感易感性增加的证据,特别是在成年人中.

科学领域:

环境健康环境健康
流行病学流行病学
公共卫生公共卫生

背景情况:

暴露于颗粒物 (PM2.5) 是对呼吸系统健康的日益关注的问题.
以前的研究表明,空气污染物与增加感染易感性之间存在联系.
了解PM2.5对流感风险的具体影响对于公共卫生干预至关重要.

研究的目的:

系统地审查和对PM2.5暴露与流感风险之间的关联进行现有研究进行元分析.
综合有关污染物影响和不同暴露时间的发现.
量化与PM2.5暴露相关的流感风险增加.

主要方法:

在PubMed,Web of Science和Scopus数据库进行系统的文献搜索,截至2010年1月1日.
遵守系统审查和元分析 (PRISMA) 准则的首选报告项目.
包括16项相关的研究用于定量分析.

主要成果:

每日PM2.5水平增加10μg/m3与流感风险上升1.5%相关 (95%CI:0.08%,2.2%).
根据暴露后的温度和滞后时间观察到风险的显著变化.
在极端温度下 (较冷:增加14.2%,较热:增加29.4%) 和在18-64岁的成年人中 (4%的增加) 发现风险更高.

结论:

暴露于PM2.5可能会损害免疫反应,导致对流感的易感性增加.
证据强烈支持PM2.5对流感风险的影响,尽管时间和气候相互作用需要进一步研究.
未来的研究应该探索不同的地区和人群,以完善公共卫生战略.

关键词:

微细颗粒物质的细颗粒物质的细颗粒物质的细颗粒物质的细颗粒物质.感染感染感染流感流感流感流感流感滞后效应滞后效应滞后效应呼吸道病毒病毒的呼吸道病毒.

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