申请长期护理保险证书的原因 对于心力衰竭患者,有缩功能障碍
Hiroki Tsuchiya1, Shinya Fujiki1, Takeshi Kashimura1
1Department of Cardiovascular and Medicine, Niigata University Graduate School of Medical and Dental Sciences, Niigata, Japan.
Geriatrics & gerontology international
|May 13, 2025
概括
在心力衰竭患者中,非心脏病是长期护理保险 (LTCI) 需求的主要原因. 了解这些原因可以改善残疾老年人的护理管理.
科学领域:
- 老年医学 老年医学
- 心脏病学 心脏病学
- 公共卫生 公共卫生
背景情况:
- 日本老龄化社会面临着越来越多的心力衰竭患者,需要长期护理.
- 在残疾老年人中,长期护理保险 (LTCI) 的使用正在增加,需要了解需求驱动因素.
研究的目的:
- 为了调查长期护理保险 (LTCI) 需求的主要原因,在心力衰竭患者中减少排气分数.
- 通过确定导致LTCI资格的条件,为更好的护理管理策略提供信息.
主要方法:
- 从2011年到2016年,在日本尼加塔市的七家医院进行了一项回顾性研究.
- 分析了来自3738名心力衰竭患者的数据,其中包括减少喷射率的患者,重点是312名新符合LTCI的患者.
- 关于LTCI引入损害的原因和程度的信息从官方注册文件中收集.
主要成果:
- 近一半 (49.4%) 的LTCI需求是由于心脏病,而50.6%源于非心脏病.
- 与主要非心脏病组 (例如癌症,中风,骨科疾病) 相比,心脏病组的身体损伤不那么严重.
- 癌症组的预后比心脏组的预后更差,与中风,骨科或其他非心脏疾病不同.
结论:
- 非心脏病是心力衰竭患者LTCI资格的一个重要因素,约占所有病例的一半.
- 确定残疾和LTCI需求的具体原因对于定制有效的护理管理至关重要.
- 这项研究强调了考虑心力衰竭患者残疾多因素原因的重要性,以优化老年护理.
更多相关视频
09:20Lumped-Parameter and Finite Element Modeling of Heart Failure with Preserved Ejection Fraction
Published on: February 13, 2021
6.3K
12:45Benefits of Cardiac Resynchronization Therapy in an Asynchronous Heart Failure Model Induced by Left Bundle Branch Ablation and Rapid Pacing
Published on: December 11, 2017
10.4K
相关概念视频
01:17Pathophysiology of Heart Failure
1.4K
Heart failure (HF) is a progressive syndrome involving ventricles that leads to inadequate cardiac output. It can be classified based on location and output or ejection fraction. Ejection fraction (EF) is an essential measurement in the diagnosis and surveillance of HF. Reduced EF corresponds to systolic heart failure (HFrEF). However, HF with preserved ejection fraction (HFpEF) is becoming increasingly prevalent. Also known as diastolic HF, this form of HF is related to aging. The...
1.4K
01:26Heart Failure Drugs: Inotropic Agents
424
Positive inotropic agents are commonly used as the first line of treatment for heart failure. One such agent is digoxin, derived from the genus Digitalis, which has been known for centuries but effectively utilized since 1785. However, these cardiac glycosides can have potentially toxic effects due to their mechanism of action, which involves inhibiting Na+/K+-ATPase and increasing contractility. Digoxin is absorbed orally and distributed in various tissues, including the CNS. It has a long...
424
01:29Documentation in Long-Term and Home Healthcare Setting
826
Documentation in long-term care facilities and home healthcare settings is crucial for ensuring continuous, coordinated, and comprehensive care for patients. Each setting has its specific documentation processes and tools:
Long-Term Care Facilities
Long-Term Care Facilities
826
01:26Heart Failure Drugs: Inhibitors of Renin-Angiotensin System
321
The activation of the sympathetic nervous system and the renin-angiotensin-aldosterone system (RAAS) contributes to cardiac remodeling, and inhibiting the RAAS is a pharmacological target in heart failure management. As a result, neurohumoral modulation is a crucial treatment principle for managing heart failure. This approach involves using medications like ACE inhibitors (ACEIs), angiotensin receptor blockers (ARBs), β-blockers, mineralocorticoid receptor antagonists (MRAs), and neutral...
321
01:22Heart Failure Drugs: β-Blockers
278
β-adrenergic antagonists, commonly known as β-blockers, block the effects of sympathetic neurotransmitters such as noradrenaline (NA) and adrenaline (ADR). They have several beneficial effects in heart failure treatment. They reduce heart rate, the force of contraction, and cardiac muscle relaxation. They also slow the atrial-ventricular conduction rate and raise the threshold for arrhythmias. The concentration of β-blockers determines their effects on bronchodilation,...
278
01:22Heart Failure Drugs: Diuretics
294
Heart failure and kidney perfusion are interconnected in a complex way. Reduced renal perfusion and venous congestion are two significant factors that contribute to renal dysfunction in heart failure. The kidneys, primarily responsible for fluid balance in the body, are adversely affected due to compromised cardiac output and increased venous pressure. In response to reduced renal perfusion, the kidneys activate neurohumoral mechanisms to restore balance. However, these mechanisms can be...
294