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在COPD中,IL-17A通过PI3K/AKT/mTOR通路调节气道重塑

Ting Ding1, Shunshun Zhao2, Yanhui Gu2

  • 1Department of Respiratory and Critical Care Medicine, Affiliated Hospital of Zunyi Medical University, Guizhou, 563003, China. tingding2024@163.com.

Scientific reports
|May 13, 2025
PubMed
概括

介素-17A (IL-17A) 通过PI3K/AKT/mTOR通路抑制巨细胞自,从而促进慢性阻塞性肺病 (COPD) 的气道重塑,导致原体沉积增加和空气流量限制.

关键词:
自自是一种自的过程.慢性肺炎肺炎呼吸道改造 呼吸道改造它们的IL-17A是IL-17A.小鼠的支气管纤维细胞.

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