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抑制TRPC3诱导糖尿病皮肤纤维细胞中的肌纤维细胞分化

Gemma Toogood1, Robin Evans2, Liping Zhang1

  • 1Department of Physiology, College of Medicine, University of Tennessee Health Science Center, Memphis, TN, United States.

Frontiers in physiology
|May 15, 2025
PubMed
概括

在糖尿病纤维细胞中,暂时受体潜在的Canonical-3 (TRPC3) 升高,损害了伤口愈合. 抑制TRPC3恢复纤维细胞功能并促进愈合,为糖尿病伤口护理提供了一种新的治疗策略.

关键词:
在TGF-β信号传输中.TRPC3 TRPC3 是一个糖尿病皮肤纤维细胞肌纤维母细胞细胞.药理上的抑制药理上的抑制.

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