在促进增殖的信号传递中对美林-Rac对抗性的形态控制

Byron G Weiss1,2, Justine M Keth1,2, Kushal Bhatt1,2

  • 1Lyda Hill Department of Bioinformatics, University of Texas Southwestern Medical Center, Dallas, TX 75390, USA.

Science signaling
|May 20, 2025
PubMed
概括

具有过度活跃的Rac1 (一种小的GTPase) 的黑色素瘤细胞使瘤抑制剂默林失活,通过增强称为lamellipodia的细胞投射来促进侵略性癌症.

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