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PLAC8作为心肌梗塞的潜在治疗标:解开分子机制

Yifan Tong1, Xin Huang2, Wei Qian3

  • 1Department of Cardiology, The First Affiliated Hospital, Jiangxi Medical College, Nanchang University, 17 Yongwai Zhengjie, Nanchang, Jiangxi Province, 330006, China. ndyfy09574@ncu.edu.cn.

Journal of physiology and biochemistry
|June 5, 2025
PubMed
概括

在心肌梗塞 (MI) 模型中,PLAC8基因表达减少,导致心肌细胞亡增加. 过度表达PLAC8抑制MEK/ERK通路,为MI提供了潜在的治疗策略.

关键词:
细胞灭亡 (apoptosis) 是一种死亡的过程.氧气过低是因为缺氧.梅克/埃尔克 (MEK/ERK) 是一个国家.心肌梗塞的心脏病发作在 PLAC8 中,

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