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Updated: Jun 13, 2025

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缺少TSC2的巨细胞具有mTORC1依赖的GPNMB增强,改善心脏缺血-再输液损伤.

Mohammad Keykhaei, Navid Koleini, Mariam Meddeb

    bioRxiv : the preprint server for biology
    |June 12, 2025
    PubMed
    概括

    通过TSC2删除在巨细胞 (MΦ) 中向拉巴胺素 (mTOR) 的机械向保护心脏免受缺血-再输液 (I/R) 损伤. 这种方法可以减少炎症并增强GPNMB蛋白,从而保持心脏功能.

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