NF-κB和PLAC8之间的相互作用损害了自,为由转换的前列腺细胞提供了生存优势

Vaibhav Shukla1, Ashish Tyagi1, Balaji Chandrasekaran1

  • 1College of Pharmacy, Texas A&M University, College Station, TX, USA.

Science advances
|June 13, 2025
PubMed
概括

暴露会通过破坏细胞死亡途径促进前列腺癌. 该研究确定NF-κB-PLAC8-BCL-xL信号轴是金属诱导前列腺癌的关键驱动因素和潜在治疗标.

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